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Age (Dordr) 2011;33:247260

Source: ScienceDirect abstract / PDF Review summarizing skin remodeling & regenerative effects of GHK/GHK-Cu

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FOXO4-DRI causes nuclear exclusion of active p53 and induces apoptosis in senescent TM3 Leydig cells Immunofluorescent staining showed elevated levels of Ser15-phospho-p53 in the nucleus of senescent TM3 Leydig cells (Figure 5A)

Over the past two decades, research (predominantly in animal models) has expanded to explore its effects on tendon, muscle, bone, nerve, and vascular tissue

GHK-Cu is a naturally occurring copper-binding tripeptide (glycyl-L-histidyl-L-lysine complexed with Cu(II)) whose endogenous serum levels decline measurably with age