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The different pools of cysteine in the cell and within the body, and their putative use as prognostic cancer markers will be also addressed

Chronic ethanol exposure not only induces hepatocyte death but also suppresses regenerative proliferation, thereby creating a pathological microenvironment conducive to ALD progression [43]

This discrepancy may highlight the critical difference between FABP2 expression in damaged enterocytes and its subsequent leakage into circulation

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