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It significantly increased the expression of Slc7a11 in the aortic intima, with attendant reduction of atherosclerosis (Figure 7K)

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F., St Jacques, R., Owens, F

High maximal and spare respiratory capacity in seal cells after hypoxia exposure occurred in concert with increasing mitochondrial branch length and independent from major changes in extracellular acidification rate, suggesting seal cells recover oxidative metabolism without significant glycolytic dependency after hypoxia exposure

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The panel of AdA and Asp performed well in identifying DILI patients with a high risk of chronicity at the early stage of the disease, which could help with early intervention in clinical practice