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Damaged AECs release pro-inflammatory and pro-fibrotic mediators, including interleukin (IL) -1 and tumor necrosis factor (TNF) -, which promote fibroblast recruitment, inflammatory cell activation (e.g., neutrophils, Th17, gdT cells), and myofibroblast differentiation, ultimately leading to ECM accumulation and fibrotic tissue remodeling [20, 29, 33, 34]

In such a condition, myomectomy, UAE, or FUS will be an alternative option for women who wish to maintain their uterus intact

The same results were corroborated in the RSL3 model, confirming the mechanism by which theaflavins inhibit cellular ferroptosis by inhibiting HO-1 (Fig

TB-500 and BPC-157 are frequently referenced together in laboratory research settings due to their complementary research pathways

mediates many of the systemic effects of RA, including the development of C-reactive protein (CRP) (Yusof & Emery, 2013)

4547 However, recent investigations have raised questions about the exclusive role of pyroptosis in APAP-induced hepatocyte loss